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Low-intensity transcranial focused ultrasound amygdala neuromodulation: a double-blind sham-controlled target engagement study and unblinded single-arm clinical trial

Bryan R. Barksdale, Lauren Enten, Annamarie DeMarco, Rachel Kline, Manoj K. Doss, Charles B. Nemeroff, Gregory A. Fonzo

Molecular Psychiatry 2025, 30, 4497-4511 · 10.1038/s41380-025-03033-w

human healthyhuman patienthealthydepressionanxietyotherfmribehaviourclinical scale

Abstract

Mood, anxiety, and trauma-related disorders (MATRDs) are highly prevalent and comorbid. A sizable number of patients do not respond to first-line treatments. Non-invasive neuromodulation is a second-line treatment approach, but current methods rely on cortical targets to indirectly modulate subcortical structures, e.g., the amygdala, implicated in MATRDs. Low-intensity transcranial focused ultrasound (tFUS) is a non-invasive technique for direct subcortical neuromodulation, but its safety, feasibility, and promise as a potential treatment is largely unknown. In a target engagement study, magnetic resonance imaging (MRI)-guided tFUS to the left amygdala was administered during functional MRI (tFUS/fMRI) to test for acute modulation of blood oxygenation level dependent (BOLD) signal in a double-blind, within-subject, sham-controlled design in patients with MATRDs (N = 29) and healthy comparison subjects (N = 23). In an unblinded treatment trial, the same patients then underwent 3-week daily (15 sessions) MRI-guided repetitive tFUS (rtFUS) to the left amygdala to examine safety, feasibility, symptom change, and change in amygdala reactivity to emotional faces. Active vs. sham tFUS/fMRI reduced, on average, left amygdala BOLD signal and produced patient-related differences in hippocampal and insular responses. rtFUS was well-tolerated with no serious adverse events. There were significant reductions on the primary outcome (Mood and Anxiety Symptom Questionnaire General Distress subscale; p = 0.001, Cohen's d = 0.77), secondary outcomes (Cohen's d of 0.43-1.50), and amygdala activation to emotional stimuli. Findings provide initial evidence of tFUS capability to modulate amygdala function, rtFUS safety and feasibility in MATRDs, and motivate double-blind randomized controlled trials to examine efficacy.ClinicalTrials.gov registration: NCT05228964.

Abstract via europepmc.

Specieshuman
Subjects52 participants
Sessions per subject1, 15swept
Randomisednot reported
Blindingdouble
Sham / controlinactive transducer
Auditory controlnot reported
Readout timingboth
Anaesthesianot applicable
Readoutsfmri, behaviour, clinical scaleMood and Anxiety Symptom Questionnaire General Distress subscale (MASQ-GD) and other MASQ subscales; PHQ-9; GAD-7; PCL-5; QIDS; STAI; BDI-II; BAI; emotional face matching task fMRI
Direction of effectinhibitoryActive vs sham tFUS/fMRI reduced left amygdala BOLD signal on average, and repeated tFUS treatment produced sustained decreases in task-based amygdala activation and clinical symptom scores.
Adverse eventsobserved24 mild-to-moderate AEs (e.g., decreased concentration, tingling, headache, dizziness) reported across 29 participants, evenly split between active and sham conditions; no serious AEs or AEs requiring medical intervention.

Exposures

Exposure 1: tFUS/rtFUS to left amygdala

Target: amygdala — “left amygdala
Device: BrainSonix · Brainsonix · Pulsar 1002

Pulse timing
Waveformpulsed
Fundamental frequency (kHz)not reported
Pulse duration (ms)5✓✓
Pulse repetition frequency (Hz)10✓✓
Duty cycle (%)5pulse duration × PRF gives 5%✓✓
Sonication duration (s)30✓✓
Pressure and intensity, by domain
Free-field pressure (kPa)not reported
Free-field Isppa (W/cm²)not reported
Free-field Ispta (W/cm²)not reported
In-situ estimatederatingsingle value
In-situ pressure (kPa)640✓✓
In-situ Isppa (W/cm²)14.4✓✓
In-situ Ispta (W/cm²)0.72✓✓
Protocol, in the paper’s words

Participants received tFUS with the following parameters: 10 Hz pulse repetition frequency (PRF), 5% duty cycle (DC), 5 ms pulse width, derated spatial peak pulse average intensity of 14.4 watts/cm2, derated spatial peak temporal average intensity of 719.91 milliwatts/cm2, and derated instantaneous peak pressure of 0.64 megapascals over 10 min in 10 sets of 30 s on/off blocks. Patients and HCs first underwent two double-blind administrations of tFUS/fMRI (active and sham order counterbalanced) separated by about a week; patients then commenced once-daily rtFUS treatment delivered 5 days a week for 3 consecutive weeks (15 sessions) using the same protocol.

Flags from extraction

  • n_subjects52 individuals (29 MATRD patients, 23 healthy controls) were enrolled and underwent tFUS/fMRI targeting; only 47 completed both active and sham sessions, so the exact number who received at least one active-condition sonication is not separately stated.
  • n_sessions_per_subjectSession count differs by subgroup: all participants (patients and healthy controls) underwent 2 tFUS/fMRI sessions (active+sham, counterbalanced), while only MATRD patients went on to receive up to 15 additional daily rtFUS treatment sessions; recorded as a list of the two distinct session counts stated in the text rather than a single combined number.
  • randomisedActive/sham order was 'counterbalanced' across participants; the paper does not state whether this counterbalancing was achieved via a randomisation procedure.
  • blindingThe target-engagement tFUS/fMRI phase was double-blind and sham-controlled, but the subsequent repetitive tFUS (rtFUS) clinical trial phase was explicitly unblinded/single-arm; 'double' reflects the sham-controlled comparison only.
  • fundamental_frequency_khzPaper does not state the fundamental/carrier ultrasound frequency of the Brainsonix Pulsar 1002 device used.