← Explore

Non-ablative disease-modifying effects of magnetic resonance-guided focused ultrasound in neuromelanin-producing parkinsonian rodents

Joan Compte, Marion Tible, Thais Cuadros, Jordi Romero-Gimenez, Ariadna Laguna, Jean-François Aubry, Erik Dumont, Charlotte Constans, Thomas Tiennot, Mathieu D. Santin, Stephane Lehericy, Miquel Vila

2023 · 10.1101/2023.08.08.552410

rodentparkinsons diseasebehaviourhistology molecular

Abstract

Age-dependent accumulation of the brain pigment neuromelanin has been implicated in the pathogenesis of Parkinson’s disease (PD). In humans, intracellular and extracellular neuromelanin levels are increased in PD postmortem brains and boosting neuromelanin production in rodents compromises neuronal function and viability and triggers a PD-like phenotype. Focused ultrasound has been shown to reduce ultraviolet light-induced skin hyperpigmentation in guinea pig and to remove brain extracellular β-amyloid plaques in Alzheimer’s mouse models. Here we show that repeated application of transcranial focused ultrasound (tFUS) is able to decrease intracellular and extracellular neuromelanin levels in neuromelanin-producing parkinsonian rats, compared to sham-treated animals, without the need for any additional therapeutic agent or intervention. Reduced neuromelanin levels in tFUS-treated animals were associated with decreased Lewy-like pathology, preserved dopaminergic phenotype, attenuated nigrostriatal degeneration, reduced glial activation, and long-term recovery of motor function. Our findings indicate that tFUS treatment applied at prodromal/early disease stages provides by itself extended structural and functional preservation of the nigrostriatal pathway in neuromelanin-producing parkinsonian rats without causing overt neuronal damage. This FDA-approved technology should thus be explored further as a noninvasive method with neuroprotective potential in PD and to maintain neuromelanin to levels below its pathogenic threshold within the aging population. One Sentence Summary Transcranial focused ultrasound reduces age-dependent neuromelanin accumulation and provides therapeutic benefit in parkinsonian rats

Abstract via europepmc.

Speciesrat (Sprague-Dawley)
Subjects6, 6swept animals
Sessions per subject3
Randomisedyes
Blindingsingle
Sham / controlundescribed
Auditory controlnot reported
Readout timingoffline
Anaesthesiaanaesthetised
Readoutsbehaviour, histology molecularCylinder test for contralateral forepaw use; stereological cell counts of TH-positive SN neurons; optical densitometry of intracellular/extracellular neuromelanin; immunohistochemistry for TH, Iba-1, CD68, GFAP and p62
Direction of effectnot assessedThis study assessed disease-modifying/neuroprotective effects (reduced intracellular/extracellular neuromelanin, attenuated nigrostriatal degeneration, improved contralateral forepaw use) rather than acute excitatory or inhibitory neuromodulation; no measure of neural excitability was reported.
Adverse eventsnone observedNo inflammatory traces were observed in the SN of tFUS-treated non-melanized (AAV-EV) control animals, indicating tFUS per se did not induce brain inflammation; thermal simulations for the parameters used estimated a maximum thermal rise of 0.12°C.

Exposures

Exposure 1: tFUS to the substantia nigra (SN) in neuromelanin-producing parkinsonian rats

Target: substantia nigra — “SN (substantia nigra), ipsilateral to AAV-TYR injection
Device: IGT / Imasonic · Imasonic (transducer); Image Guided Therapy (IGT), Pessac, France (Thermoguide guidance system)

Pulse timing
Waveformpulsed
Fundamental frequency (kHz)1,500✓✓
Pulse duration (ms)3✓✓
Pulse repetition frequency (Hz)10.31✓✓
Duty cycle (%)not reportedpulse duration × PRF gives 3.093%
Sonication duration (s)600✓✓
Pressure and intensity, by domain
Free-field pressure (kPa)1,050✓✓
Free-field Isppa (W/cm²)not reported
Free-field Ispta (W/cm²)1.1✓✓
In-situ estimatenot reported
In-situ pressure (kPa)not reported
In-situ Isppa (W/cm²)not reported
In-situ Ispta (W/cm²)not reported
Protocol, in the paper’s words

MRI-guided tFUS/sham treatments were performed once a week for 3 consecutive weeks, targeting the SN ipsilateral to AAV injections. The power of the tFUS ultrasound beam was initially set at 5% corresponding to a Peak Negative Pressure of 1.05 MPa in water according to the calibration, with a duration of 3 ms every 97 ms for 600 repetitions (10 minutes), corresponding to Ispta=1.1 W/cm2. Behavioural/histological effects were assessed at 4 months post-AAV injection, ~2.5 months after the last tFUS application.

Flags from extraction

  • exposures[0].timing.pulse_repetition_frequency_hzPRF was not stated directly; converted from the stated inter-pulse period ('a duration of 3 ms every 97 ms') to ~10.3 Hz, a unit conversion rather than an inference.
  • sham_typeThe paper contrasts 'tFUS' vs 'sham' treatment groups but never describes what the sham procedure physically involved (transducer powered off, blocked, aimed away, etc.).
  • n_subjectsOnly the two constituent tFUS-treated group sizes (EV:tFUS n=6, TYR:tFUS n=6) are stated; the paper never states a combined total, so both group sizes are listed rather than summed.
  • blindingBlinding is stated only for outcome assessors ('investigator blinded to the experimental groups') during behavioural/histological quantification; no statement is made about blinding of the individuals administering tFUS/sham treatment.