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Ultrasound Used for Diagnostic Imaging Facilitates Dendritic Branching of Developing Neurons in the Mouse Cortex

Tamas Papp, Zsuzsanna Ferenczi, Bernadette Szilagyi, Matyas Petro, Angelika Varga, Eva Kókai, Ervin Berenyi, Gabor Olah, Gabor Halmos, Peter Szucs, Zoltan Meszar

Frontiers in Neuroscience 2022, 16 · 10.3389/fnins.2022.803356

rodenthealthycellular imaginghistology molecular

Abstract

Neuronal differentiation and synaptogenesis are regulated by precise orchestration of intrinsic and extrinsic chemical and mechanical factors throughout all developmental steps critical for the assembly of neurons into functional circuits. While ultrasound is known to alter neuronal migration and activity acutely, its chronic effect on neuronal behavior or morphology is not well characterized. Furthermore, higher-frequency (3-5 MHz) ultrasound (HFU) is extensively used in gynecological practice for imaging, and while it has not been shown harmful for the developing brain, it might be associated with mild alterations that may have functional consequences. To shed light on the neurobiological effects of HFU on the developing brain, we examined cortical pyramidal cell morphology in a transgenic mouse model, following a single and short dose of high-frequency ultrasound. Layer V neurons in the retrosplenial cortex of mouse embryos were labeled with green and red fluorescent proteins by in utero electroporation at the time of their appearance (E14.5). At the time of their presumptive arrival to layer V (E18.5), HFU stimulation was performed with parameters matched to those used in human prenatal examinations. On the third postnatal day (P3), basic morphometric analyses were performed on labeled neurons reconstructed with Neurolucida. Low-intensity HFU-treated cells showed significantly increased dendritic branching compared to control (non-stimulated) neurons and showed elevated c-fos immunoreactivity. Labeled neurons were immunopositive for the mechanosensitive receptor TRPC4 at E18.5, suggesting the role of this receptor and the associated signaling pathways in the effects of HFU stimulation.

Abstract via europepmc.

Speciesmouse (CD1/ICR)
Subjects24, 9swept animals
Sessions per subject1, 5swept
Randomisednot reported
Blindingnot reported
Sham / controlinactive transducer
Auditory controlnot reported
Readout timingoffline
Anaesthesiaanaesthetised
Readoutscellular imaging, histology molecularNeurolucida 3D dendritic reconstruction/morphometry of GFP- and Brainbow-labeled layer V pyramidal neurons; c-Fos, BDNF and TRPC4 immunohistochemistry; BDNF qRT-PCR
Direction of effectexcitatoryA single prenatal ultrasound exposure increased the number of basal dendrites and elevated c-Fos immunoreactivity (a marker of neuronal activation) in retrosplenial layer V pyramidal neurons; a single exposure did not change BDNF, but repeated (5x) weekly exposure additionally increased BDNF immunoreactivity.
Adverse eventsnot reported

Exposures

Exposure 1: Diagnostic ultrasound exposure of pregnant mice / pups (retrosplenial cortex neurons analysed)

Target: retrosplenial cortex — “retrosplenial cortex
Device: other named manufacturer · GE · Logiq V2 (4C-RS convex probe)

Pulse timing
Waveformnot reported
Fundamental frequency (kHz)3,000✓✓
Pulse duration (ms)not reported
Pulse repetition frequency (Hz)not reported
Duty cycle (%)not reported
Sonication duration (s)600✓✓
Pressure and intensity, by domain
Free-field pressure (kPa)not reported
Free-field Isppa (W/cm²)not reported
Free-field Ispta (W/cm²)not reported
In-situ estimatenot reported
In-situ pressure (kPa)not reported
In-situ Isppa (W/cm²)not reported
In-situ Ispta (W/cm²)not reported
Pressure, domain unspecified (kPa)1,557✓✓
Ispta, domain unspecified (W/cm²)0.056✓✓
Protocol, in the paper’s words

Mice previously subjected to in utero electroporation were deeply anesthetized and received a single 10-minute abdominal diagnostic ultrasound exposure (GE Logiq V2, 4C-RS convex probe, 3 MHz, MI=0.9, TIS=0.8) at embryonic day 18.5 (E18.5); the probe covered the whole abdominal region so all embryos received the same dose. After birth, unfocused ultrasound was applied directly to the skull. In a separate cohort, the E18.5 exposure was followed by 4 further weekly ultrasound stimuli during the first 4 postnatal weeks (5 exposures total). Sham/control animals underwent identical handling with the transducer pressed to the abdomen but without actual ultrasound output.

Flags from extraction

  • exposures[0].target.termsUltrasound was applied to the whole maternal abdomen (unfocused with respect to the fetal brain); retrosplenial cortex was the region subsequently analysed for morphological effects, not a spatially targeted stimulation site.
  • exposures[0].unspecified_domain.ispta_w_cm2Paper states a single 'calculated average intensity' (56 mW/cm2) without specifying whether this is spatial-peak pulse-average (Isppa) or spatial-peak temporal-average (Ispta) intensity, or the domain (water/free-field vs in situ); recorded as Ispta by convention and flagged.
  • exposures[0].unspecified_domain.pressure_kpaDomain (free-field vs in situ) not stated by the paper for this 'calculated peak negative acoustic pressure'; placed in unspecified_domain per the three-way rule.
  • n_subjectsPaper gives separate group sizes for the electroporation/morphometry cohort (24 embryos enrolled, real US) and a separate qRT-PCR cohort (9 pups, real US); recorded as a list per SCHEMA.md guidance. An additional 'five times repetitive' subgroup exists but its own N is not stated, so it is not included in this list.
  • n_sessions_per_subjectMost animals received a single 10-min exposure at E18.5, but a separate repetitive-exposure subgroup received 5 total exposures (E18.5 plus 4 further weekly doses); no single value applies to the whole study.
  • adverse_eventsPaper reports no gross morphological changes in most dendritic parameters and intact cortical layering, but does not make an explicit overall safety/adverse-event statement for the animals or fetuses.